This study investigated whether γ-T3 suppresses human cervical cancer HeLa cell growth through modulation of the phosphatidylinositol 3-kinase/protein kinase B/mammalian target of rapamycin (PI3K/AKT/mTOR) signaling pathway.<h4>Methods</h4>HeLa cells were treated with γ-T3 at different concentrations (0-80 μmol/L). The gene discussed is MTOR; the disease is cervical cancer.