Dominant mechanistic categories included activation of innate and adaptive immunity and reversal of tumor microenvironment immunosuppression (notably Th1-driven IL-12/IFN-γ responses, antitumor M1 macrophage polarization), induction of apoptosis, anti-angiogenesis, molecular mimicry and modulation of cancer-pertinent pathways. Here, IFNG is linked to neoplasm.