When we divided the population by patient/control status, we found that, after adjustment for all investigated variables (HbA1c, GFR, CRP, copeptin, age, smoking, cortisone treatment, physical activity, lipid-lowering medication, and gender), both ApoA1 (HR 4.290, CI 1.871-9.837, <i>p</i>-value < 0.001) and ApoB (HR 7.625, CI 1.995-29.138, <i>p</i>-value 0.003) remained independently associated with fracture risk in the T1D group. The gene discussed is APOA1; the disease is type 1 diabetes mellitus.