We reveal many novel adaptive loci with convergent signals from selection, infectious disease GWAS and immune-gene QTLs (including at <i>FUT6</i> for intestinal infections; at <i>ASAP1</i> for TB; and at <i>LYZ</i>, an antimicrobial enzyme), fine-mapping selection onto likely causal variants. This evidence concerns the gene ASAP1 and tuberculosis.