BRAF and neoplasm: Molecular analyses revealed two main families of drivers for PTC ‐ BRAF V600E or BRAF V600E–like mutations which aligned with “classical papillary nucleus”, papillary architecture, and conventional PTC outcomes, and RAS or RAS–like mutations that were associated with “papillary‐like nucleus”, follicular architecture, and follicular‐patterned neoplasm outcomes.