Similar deletions have also been observed in patients with epilepsy, indicating that <i>Alg13</i> may play a critical role in cortical interneuron development.<h4>Methods</h4>Immunofluorescence analysis was used to assess the effects of <i>Alg13</i> deletion on the distribution and migration of interneurons in the cerebral cortex of postnatal mice. The gene discussed is ALG13; the disease is epilepsy.