In an LPS-induced ARDS mouse model, DG treatment not only significantly reduced serum levels of inflammatory cytokines but also increased the activity of the antioxidant enzyme superoxide dismutase (SOD), decreased the levels of myeloperoxidase (MPO) and malondialdehyde (MDA), and markedly alleviated pulmonary histopathological damage, demonstrating notable tissue-protective effects. The gene discussed is MPO; the disease is acute respiratory distress syndrome.