Specifically, we evaluated its ability to induce immunogenic cell death (ICD), activate dendritic cells (DCs) via the cGAS-STING signaling pathway, and influence CD8<sup>+</sup> T cell recruitment and infiltration within the tumor microenvironment.<h4>Results</h4>Treatment with nab-PTX induced ICD in TNBC cells was associated with enhanced activation of DCs through the cGAS-STING pathway. This evidence concerns the gene STING1 and neoplasm.