However, clinical application is limited by the absence of a standardized cutoff, lack of assay harmonization, and variability across PCOS phenotypes.<h4>Objective</h4>To compare AMH levels across the 4 PCOS phenotypes (A, B, C, D) defined by the Rotterdam criteria.<h4>Data sources</h4>PubMed, Embase, ScienceDirect, and Web of Science were searched for studies published between January 2009 and July 2024. This evidence concerns the gene AMH and polycystic ovary syndrome.