TP53 and cervical carcinoma: Simultaneously, it triggered ferroptosis via ROS accumulation, GSH depletion, elevated MDA and Fe<sup>2+</sup>, and suppression of SLC7A11/GPX4.<h4>Discussion</h4>B1 is a promising dual-mechanism lead compound against cervical cancer, concurrently activating p53 and ferroptosis, warranting further investigation.