Laboratory examination showed severe autoimmune hypothyroidism with a thyroid-stimulating hormone (TSH) level of >100 μIU/mL, significantly low free T4 and T3 concentrations, positive results for anti-thyroid peroxidase and anti-thyroglobulin antibodies, and elevated creatine kinase indicative of rhabdomyolysis in the setting of concurrent acute kidney injury (AKI). This evidence concerns the gene TG and Hashimoto thyroiditis.