Neonatal exposure induced cerebellar hypoplasia, Purkinje degeneration, heterotopic gray matter, microcephaly, hydrocephalus and impaired brain growth [32,34,35,36,40,41,42,44], alongside p53-dependent fetal lesions [101], sensorimotor gating deficits [43] and neurochemical disturbances [39]. The gene discussed is TP53; the disease is Hydrocephalus.