OLR1 and Myocardial fibrosis: When hypertension is combined with dyslipidemia, LDL is oxidized to oxLDL, which invades and deposits by damaging LOX-1 on the surface of vascular endothelial cell membrane [35], inhibits eNOS, reduces NO production, damages vasomotor function, increases blood pressure and activates RAAS, and aggravates myocardial fibrosis [36,37].