AVP and Anxiety: This model emphasizes that complex behavioral syndromes can emerge from the differential recruitment of parallel, functionally specialized pathways downstream of a common hub, revealing new anatomical targets for developing symptom-specific interventions for stress-related psychopathology [25,68]—for instance, targeting BNST microcircuits receiving BLA input (e.g., CRH neurons) to alleviate anxiety, or modulating the direct PFC-input BNST pathway (e.g., AVP/OT neurons) to improve social function [68,69].