AKT1 and myocarditis: Based on the convergent evidence from enrichment analysis, molecular docking, and animal validation, we have identified Akt as the key target of LC against myocarditis: it exhibits optimal topological centrality in the PPI network, demonstrates the strongest binding affinity with LC, and serves as a signaling hub in the PI3K/Akt pathway that is closely associated with the pathological progression of myocarditis.