LEPR and familial dilated cardiomyopathy: A recent study validated that the global m6A level of heart samples was relatively higher in a DCM model with leptin receptor deficiency than in normal mice, and the MeRIP-seq results of DCM and normal hearts revealed that differentially m6A-modified genes were related to cardiac fibrosis, myocardial hypertrophy, and myocardial energy metabolism in DCM (116).