To further investigate how tumour background influences the immune microenvironment of myocardial infarction thrombi, we performed immunohistochemical analysis of coronary thrombus samples from the T1MI group and from patients with both LUAD and T1MI, assessing the expression levels of CD8, CXCL8, PTX3, PD‐L1 and FOXP3 (Figures 11a, 11b, 11c, 11d, 11e and 11f). The gene discussed is CD8A; the disease is myocardial infarction.