This expression pattern highlights a functional dichotomy: stemness-promoting genes (EIF5A, MELTF) are selectively upregulated at the protein level, whereas metabolic homeostasis genes (CPS1, SELENOK) are suppressed, reinforcing the hypothesis that OSA-associated hypoxia drives a shift toward translational and iron-regulated growth programs while diminishing metabolic resilience. This evidence concerns the gene CPS1 and obstructive sleep apnea syndrome.