Previous reports suggest that heterogeneous pathophysiology influences the composition of troponin release, with long cTnT levels higher in Takotsubo syndrome than in end-stage kidney disease or after heavy exercise, even though smaller troponin fragments were mainly responsible for troponin elevations in all these conditions.9,13,14 Moreover, we did not obtain consent from a relatively large proportion of patients with early ED discharge who were contacted by mail, but most admitted patients did consent to participate in the study. Here, TNNT2 is linked to Tako-tsubo cardiomyopathy.