These findings align with our study, which not only identified all three RV species (A, B, and C) in tonsillar tissues from children without acute symptoms, but also demonstrated active infection and replication in CD4+ T and B lymphocytes, accompanied by the release of pro‐inflammatory cytokines such as IL‐17, TNF‐α, and IFN‐γ. The gene discussed is IFNG; the disease is infection.