In in vivo experiments with BABL/c mice inoculated with U251 and U87 cells intracranially, the knockdown of MRPS16 and overexpression of NFATC2 led to the tumours in the mice exhibiting a high growth rate compared with mice inoculated with cells that only had the MRPS16 gene knocked down. This evidence concerns the gene NFATC2 and neoplasm.