The ACSL4 knockout mouse model demonstrated that, compared to the ACSL4 wild‐type, the ACSL4 knockout group exhibited reduced infiltration of CD8+ T cells within tumors, decreased expression of IFN‐γ and tumor necrosis factor alpha, lowered levels of intratumoral lipid peroxides, and significantly increased tumor volume [79]. The gene discussed is ACSL4; the disease is neoplasm.