Clinical studies have shown that WXKL may suppress sarcoplasmic reticulum Ca2+ release and maintain intracellular Ca2+ balance,[7] regulate mitochondrial function and homeostasis,[8] regulate the CaMK II signal transduction pathway to inhibit arrhythmia and improve cardiac function.[9] Accumulating evidence from various animal and cell studies has shown that WXKL could protect the myocardium and anti-arrhythmia against cardiovascular diseases. Here, CAMK2G is linked to Arrhythmia.