Herpesvirus infection does not change the expression of lamins, but results in local dispersion of the nuclear lamina by phosphorylation and redistribution of lamins A/C and B. Various viral proteins and kinases (UL13, UL34, ICP34.5, and Us3) mediate these processes [39,74–77]. The gene discussed is RPL13A; the disease is Herpesviridae infectious disease.