CD79A and acute myeloid leukemia: Our gene expression analysis concurs with previous reports, where overexpression of CD74, CTLA-4, HLA-C, CD79A, IRF5 and LAG3 has been associated with AML and other types of cancer, indicating their potential participation in tumour development, either as tumour initiators or as immunological checkpoint modulators that allow malignant progression [76,77,90–95].