Laboratory investigations revealed polymorphonuclear leukocytosis (21.14 × 109/L, 93.8% neutrophils), elevated C-reactive protein (CRP) (53.4 mg/L), hyperglycemia (13.7 mmol/L), and a markedly raised troponin I (738 ng/L), which was suspected to represent neurogenic cardiac injury secondary to the severe subarachnoid hemorrhage, further underscoring the catastrophic physiological stress of the event. Here, CRP is linked to Hyperglycemia.