For instance, in breast cancer, SNHG5 regulates proliferation through the SNHG5–miR-154-5p–PCNA axis [31]; in HCC, it sponges miR-26a-5p to relieve repression of GSK3β, leading to Wnt/β-catenin pathway activation and EMT induction [32]. This evidence concerns the gene PCNA and breast carcinoma.