Consistent with our findings for KRAS, we found that ectopically expressed NRAS phospho-mutants modulated ubiquitin binding, with increased ubiquitin binding observed for NRASG12D+T148A in both the SKMM1 MM line and THP1 AML cells (Fig. 5F, S5E). The gene discussed is KRAS; the disease is acute myeloid leukemia.