IL4 and acute respiratory distress syndrome: The liver regeneration observed in the pathological anatomy is represented by the presence of eosinophils that migrate toward hepatocytes; these recruited eosinophils secrete IL‐4 and subsequently IL‐4/IL‐13 signaling is activated which facilitates liver regeneration by stimulating the entry of inactive hepatocytes into the cell cycle, resulting in a favorable prognosis especially in patients with ALI with preserved hepatic functional reserve [10] as in the case of our patient.