Further highlighting the importance of EV subtypes, it was found that while overall EV concentration did not differ between ALS patients and controls, microvesicles in ALS showed increased size and carried pathological proteins (e.g., SOD1, TDP‐43, p‐TDP‐43, FUS), suggesting a role in prion‐like propagation (Sproviero et al. 2018). The gene discussed is SOD1; the disease is amyotrophic lateral sclerosis.