UMOD and autosomal dominant medullary cystic kidney disease with or without hyperuricemia: Although endoplasmic reticulum stress, cell damage and apoptosis induced by the accumulation of mutant uromodulin have been identified as pathogenic mechanisms in ADTKD‐UMOD [4, 5, 6, 7, 8], additional findings suggest that other mechanisms may also contribute to renal fibrosis [10].