As the most prominent tissue‐resident ILC3 subsets in tumour, ILC3‐HSPA1B is distinguished by upregulated expression of cell stress‐related genes (e.g., HSPA1B, HSPA1A, and JUN), all of which have been reported to be involved in cellular stress responses and adaptive mechanisms.56, 66. The gene discussed is HSPA1A; the disease is neoplasm.