CD36 and atrial fibrillation: Samples of left atrial myocardium from individuals with AF showed dysregulatedmetabolism compared to those in sinus rhythm, including elevated expression ofCaMKII and AMPK, increased membrane levels of fatty acid translocase/cluster ofdifferentiation 36 (FAT/CD36), lipid accumulation, reduced GLUT-4 membraneexpression, increased glycogen storage, and higher levels of the pro-apoptoticprotein Bax [144].