Breast cancer is a molecularly heterogeneous disease, with treatment strategies guided by specific molecular targets such as receptor tyrosine kinases (e.g., EGFR and HER2), hormonal receptors (ER and PR), and key regulatory proteins involved in tumor progression, including PIK3CA, CDKs, and tubulin [5, 26, 27, 55, 60]. This evidence concerns the gene EGFR and breast carcinoma.