It has been reported that CCL5 exerts its biological effects by engaging C‐C chemokine motif receptor 1 (CCR1), CCR3, and CCR5 receptors on cell surfaces, activating signaling pathways like the signal transducer and activator of transcription factor 3, nuclear factor (NF)‐κb, and mitogen‐activated protein kinase (MAPK), observed in conditions like non‐alcoholic fatty liver disease (NAFLD), atherosclerosis, and myocardial infarction (Li, Sun, et al. 2020; Montecucco et al. 2012). This evidence concerns the gene CCR3 and metabolic dysfunction-associated steatotic liver disease.