In another study, the potential mechanisms of Cyanidin-3-glucoside in lung adenocarcinoma were presented, showing that it exerts anticancer effects through suppression of TP53I3 (Tumor Protein P53 inducible Protein 3) expression and blockade of the PI3K/AKT/mTOR signaling cascade [64]. This evidence concerns the gene TP53I3 and lung adenocarcinoma.