Other interesting DEGs of note were multiple members of the solute carrier (SLC) family, including the downregulation of the Na+/K+/Ca2+ exchanger SLC24A1, which, when dysfunctional, leads to congenital stationary night blindness (CSNB) [22] and upregulation of the potassium channel interacting protein KCNIP2, which has been reported to regulate Kv4 (KCND) channels [23]. This evidence concerns the gene KCNC1 and congenital stationary night blindness.