NFKB1 and diabetic kidney disease: In DKD, miRNAs modulate a wide range of pathology pathways—fibrosis, inflammation, oxidative stress, and EMT—mainly through multiple canonical signaling pathways such as transforming growth factor-β/Smad, nuclear factor-κB (NF-κB), phosphatidylinositol-3-kinase/protein kinase B (PI3K/Akt), mitogen-activated protein kinase (MAPK), and Wnt/β-catenin [22,27].