JUN and cervical carcinoma: The GO analysis showed several associations with cell death processes, regulation of DNA replication, protein-binding regulation, and transcription factors as JUN, SP1, ELF3, E2F1, P53, RELA, HDAC, and FOXM1, which have been reported to contribute to the promotion of cervical cancer, which is also regulated by oncomiRs and tumor-suppressing microRNAs [78,79,80], which are also associated with DEGs in this work.