Differentially expressed genes in the livers of PCOS rats, compared with controls, were predominantly enriched in pathways such as steroid hormone biosynthesis, linoleic acid metabolism, taurine and hypotaurine metabolism, fatty acid degradation, and the PI3K–AKT signaling pathway—findings in agreement with earlier reports [42–45]. The gene discussed is AKT1; the disease is polycystic ovary syndrome.