Though not reaching statistical significance, the ERVs were found in the flanking region of genes, such as FANCI (FA complementation group I), associated with fanconi anemia25, and FAP (fibroblast activation protein alpha) and CX3CL1 (C-X3-C motif chemokine ligand 1), candidates for polyarteritis nodosa, a disease causing inflammation of the arteries26. The gene discussed is FAP; the disease is Friedreich ataxia.