SNAI1 and acute coronary syndrome: CXCL5 activates epithelial-to-mesenchymal transition (EMT)-related pathways (ERK/Elk-1/Snail and PI3K/AKT/GSK-3β/Snail-Twist), which may have parallels in vascular remodeling and fibrosis after myocardial injury and influencing the development of collateral circulation after an acute coronary syndrome [60].