Peng et al. showed that ZFAS1 contributed to intrinsic resistance to low-dose radiation (at 8Gy) in NPC cell models (C666-1 and SUNE-1), as specific siRNA knockdown of ZFAS1 by siRNA resulted in increased apoptosis upon radiation treatment in vitro, and such resistance could be mediated via mir-7-5p [108]. The gene discussed is ZFAS1; the disease is nasopharyngeal carcinoma.