The findings that pharmacologic inhibition of FTO with FB23-2 caused significant and dose-dependent increases in the protein levels of these fetal genes, including MYH7, ANP, and BNP, in neonatal cardiomyocytes, suggesting that FTO repression or inhibition may play a role in upregulating cardiac fetal genes and induce cardiac hypertrophy. Here, MYH7 is linked to cardiac hypertrophy.