Since the amygdaloid complex has been described as a site for multiple pathologies, immunohistochemistry against different proteinopathies (Supplementary Table 1) confirmed the copathologies of neuropathological diagnosis with the presence of tau (Fig. 4), amyloid β (Fig. 5) and TDP43 (Fig. 6) but not α-synuclein (Fig. 7). The gene discussed is TARDBP; the disease is proteostasis deficiencies.