Significantly suppresses oxidative stress and damage in cells, inhibits ANGII-induced proliferation of cardiac fibroblasts, downregulates activation of the PI3K/AKT-mTOR pathway, suppresses α-SMA expression, and promotes COL2 expression, thereby alleviating myocardial fibrosis and improving cardiac function. The gene discussed is MTOR; the disease is Myocardial fibrosis.