Moreover, CD36-mediated phagocytosis is modulated by inflammatory signals:pro-inflammatory cytokines such as tumor necrosis factor-α(TNF-α) and interleukin (IL)-1β suppress CD36 expression,delaying hematoma resolution and exacerbating neurological deficits, whereas theanti-inflammatory cytokine IL-10 enhances CD36 expression, accelerating hematomaclearance and functional recovery [27]. The gene discussed is CD36; the disease is hematoma.