Meanwhile, MARK4, a serine/threonine kinase, enhances tumor cell stress resistance by phosphorylating the Fat Mass and Obesity‐Associated Protein (FTO) under stress conditions, which subsequently promotes the hyperacute translation of Hyperacute Response Proteins (HARPs) within γ‐tubulin‐associated translation microdomains [38, 39]. Here, MARK4 is linked to neoplasm.