Each dataset was chosen for its mechanistic significance: PSPC1 (GSE247301) suggests the maintenance of leukaemic stem cells through PU.1 interaction; JMJD1C and RUNX1 (GSE251728) co‐regulate leukaemic transcriptional condensates; VEN + CSA (GSE282105) addresses metabolic vulnerabilities in FLT3‐ITD AML via PI3K/AKT/mTOR modulation. The gene discussed is MTOR; the disease is acute myeloid leukemia.