For this, we gated on non-hematopoietic cells (CD45−), lacking expression of CD234a (Duffy antigen), CD31 (endothelial cells), EPCAM (epithelial cells), and CD34 (progenitors) but expressing the fibroblast marker CD90, as well as PDPN and FAP, which are both canonical myofibroblast markers and hubs genes in our human TB-myofibroblast module (Fig. 6 A). This evidence concerns the gene THY1 and tuberculosis.